World Library  
Flag as Inappropriate
Email this Article


Article Id: WHEBN0014543697
Reproduction Date:

Title: Cxcr6  
Author: World Heritage Encyclopedia
Language: English
Subject: CCR8 (gene), C-C chemokine receptor type 6, Interleukin 5 receptor alpha subunit, Interleukin 8 receptor, alpha, CXCR3
Publisher: World Heritage Encyclopedia


Chemokine (C-X-C motif) receptor 6
Symbols  ; BONZO; CD186; STRL33; TYMSTR
External IDs IUPHAR: ChEMBL: GeneCards:
RNA expression pattern
Species Human Mouse
RefSeq (mRNA)
RefSeq (protein)
Location (UCSC)
PubMed search

C-X-C chemokine receptor type 6 is a protein that in humans is encoded by the CXCR6 gene.[1][2][3]

CXCR6 has also recently been designated CD186 (cluster of differentiation 186).


  1. ^ Liao F, Alkhatib G, Peden KW, Sharma G, Berger EA, Farber JM (Jun 1997). "STRL33, A novel chemokine receptor-like protein, functions as a fusion cofactor for both macrophage-tropic and T cell line-tropic HIV-1". J Exp Med 185 (11): 2015–23.  
  2. ^ Deng HK, Unutmaz D, KewalRamani VN, Littman DR (Aug 1997). "Expression cloning of new receptors used by simian and human immunodeficiency viruses". Nature 388 (6639): 296–300.  
  3. ^ "Entrez Gene: CXCR6 chemokine (C-X-C motif) receptor 6". 

External links

  • "Chemokine Receptors: CXCR6". IUPHAR Database of Receptors and Ion Channels. International Union of Basic and Clinical Pharmacology. 

Further reading

This article incorporates text from the United States National Library of Medicine, which is in the public domain.

This article was sourced from Creative Commons Attribution-ShareAlike License; additional terms may apply. World Heritage Encyclopedia content is assembled from numerous content providers, Open Access Publishing, and in compliance with The Fair Access to Science and Technology Research Act (FASTR), Wikimedia Foundation, Inc., Public Library of Science, The Encyclopedia of Life, Open Book Publishers (OBP), PubMed, U.S. National Library of Medicine, National Center for Biotechnology Information, U.S. National Library of Medicine, National Institutes of Health (NIH), U.S. Department of Health & Human Services, and, which sources content from all federal, state, local, tribal, and territorial government publication portals (.gov, .mil, .edu). Funding for and content contributors is made possible from the U.S. Congress, E-Government Act of 2002.
Crowd sourced content that is contributed to World Heritage Encyclopedia is peer reviewed and edited by our editorial staff to ensure quality scholarly research articles.
By using this site, you agree to the Terms of Use and Privacy Policy. World Heritage Encyclopedia™ is a registered trademark of the World Public Library Association, a non-profit organization.

Copyright © World Library Foundation. All rights reserved. eBooks from Project Gutenberg are sponsored by the World Library Foundation,
a 501c(4) Member's Support Non-Profit Organization, and is NOT affiliated with any governmental agency or department.